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Rapid Communication | Open Access

Neutrophil extracellular traps induced by interleukin 8 via CXCR1/2 promote the progression of gastric carcinoma through transcription factor IIB-related factor 1 and cyclin

Qianling Wanga,1Yiyin Zhanga,1Wenxi Dinga,1Cheng FengbYuyan WangaXiaoli WeiaZiting QuaHui WangaXiaoying LiubHua Wanga( )Kangsheng Gua( )
Department of Oncology, The First Affiliated Hospital of Anhui Medical University, Hefei, Anhui 230022, China
School of Life Sciences, Anhui Medical University, Hefei, Anhui 230032, China

1 These authors contributed equally to this work.

Peer review under responsibility of Chongqing Medical University.

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References

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Zhang Y, Wu H, Yang F, et al. Prognostic value of the expression of DNA repair-related biomarkers mediated by alcohol in gastric cancer patients. Am J Pathol. 2018;188(2):367–377.

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Zhang Y, Hu Y, Ma C, et al. Diagnostic, therapeutic predictive, and prognostic value of neutrophil extracellular traps in patients with gastric adenocarcinoma. Front Oncol. 2020;10:1036.

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Wang H, Zhang Y, Wang Q, et al. The regulatory mechanism of neutrophil extracellular traps in cancer biological behavior. Cell Biosci. 2021;11:193.

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Nie M, Yang L, Bi X, et al. Neutrophil extracellular traps induced by IL8 promote diffuse large B-cell lymphoma progression via the TLR9 signaling. Clin Cancer Res. 2019;25(6):1867–1879.

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Loveridge CJ, Slater S, Campbell KJ, et al. BRF1 accelerates prostate tumourigenesis and perturbs immune infiltration. Oncogene. 2020;39(8):1797–1806.

Genes & Diseases
Pages 575-578
Cite this article:
Wang Q, Zhang Y, Ding W, et al. Neutrophil extracellular traps induced by interleukin 8 via CXCR1/2 promote the progression of gastric carcinoma through transcription factor IIB-related factor 1 and cyclin. Genes & Diseases, 2024, 11(2): 575-578. https://doi.org/10.1016/j.gendis.2023.03.025

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Received: 22 December 2022
Published: 26 April 2023
© 2023 The Authors.

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

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